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Hydrogen peroxide for plants9/20/2023 ![]() ![]() ![]() Though changes in size and growth-related parameters are normal and expected, plants lack several functional abilities as a direct consequence of their sedentary lifestyles (De-Bruxelles and Roberts 2001). The lifespan and developmental timeframe of many plant species are characterized by a state of limited mobility. Avenues of future research are also considered, including the identification of factors with differential activity in abiotic stress versus wound-induced response pathways, applications to pest control, and creation of quantitative models for peroxide activity. Much of the current work on this topic has focused on uncovering novel sources of H 2O 2 in response pathways, examining overarching molecular networks that control oxidative burst events, assessing interactions between various environmental and endogenous factors and H 2O 2 signaling, and studying gene products that protect cellular components during H 2O 2 responses. Hydrogen peroxide is also one of the earliest signaling molecules involved in wound-signaling pathways, serving as a local signal for hypersensitive cell death and cell wall stiffening events, and as a mobile signal that stimulates defense-genes in adjacent cells (Orozco-Cardenas et al. Majority of the early research linking H 2O 2 with specific plant responses to the environment dealt with responses to abiotic stressors, which are dependent on peroxide accumulation mediated by calcium ion movement and mitogen-activated protein kinase cascades. Hydrogen peroxide (H 2O 2) is an endogenous molecule that is part of a group of cellular components referred to as reactive oxygen species (ROS) that are formed by aerobic respiration and other oxidation-related processes within the plant (Orozco-Cardenas, et al. The major purpose of this review article is to explore the influence of hydrogen peroxide on cell signaling and gene expression patterns in plants, within the context of defense responses to a variety of biotic stressors, abiotic stressors, and wounding events. ![]()
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